oligo 2 Search Results


rabbit anti olig2 antibody  (Proteintech)
96
Proteintech rabbit anti olig2 antibody
Aurkb deficiency in microglia disrupts remyelination and oligodendrocyte density in the CPZ-induced demyelination model (A) Adult Aurkb fl/fl and Cx3cr1 CreERT2/+ Aurkb fl/fl mice (2–3 months old) were i.p. injected with TAM for 5 consecutive days. After one month, the mice were fed a 0.25% CPZ-enriched diet for 5 weeks to induce demyelination (CPZ group) or followed by a CPZ-free normal diet for an additional 2 weeks (Recovery group). The corpus callosum (CC) was analyzed by immunofluorescence (IF) and Black-Gold II myelin staining. (B and C) Representative Black-Gold II myelin staining images and quantification of myelin intensity ( n = 5 mice per genotype per group, Scale bars: 200 μm). (D–F) Representative immunofluorescence analysis and quantification of total Mbp and oligodendrocytes <t>(Olig2</t> + ), and g-h) representative immunofluorescence analysis and quantification of mature oligodendrocytes (CC-1 + ) ( n = 5 mice per genotype per group, Scale bars: 50 μm). Data are presented as the mean ± SD. two-way ANOVA with Bonferroni multiple comparisons test in (C, E, F, and H); ∗∗ p < 0.01 and ∗∗∗ p < 0.001; ns, not significant compared with the Aurkb fl/fl group.
Rabbit Anti Olig2 Antibody, supplied by Proteintech, used in various techniques. Bioz Stars score: 96/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/result/rabbit anti olig2 antibody/product/Proteintech
Average 96 stars, based on 1 article reviews
rabbit anti olig2 antibody - by Bioz Stars, 2026-04
96/100 stars
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anti oligodendrocyte transcription factor 1  (Rockland Immunochemicals)
86
Rockland Immunochemicals anti oligodendrocyte transcription factor 1
Aurkb deficiency in microglia disrupts remyelination and oligodendrocyte density in the CPZ-induced demyelination model (A) Adult Aurkb fl/fl and Cx3cr1 CreERT2/+ Aurkb fl/fl mice (2–3 months old) were i.p. injected with TAM for 5 consecutive days. After one month, the mice were fed a 0.25% CPZ-enriched diet for 5 weeks to induce demyelination (CPZ group) or followed by a CPZ-free normal diet for an additional 2 weeks (Recovery group). The corpus callosum (CC) was analyzed by immunofluorescence (IF) and Black-Gold II myelin staining. (B and C) Representative Black-Gold II myelin staining images and quantification of myelin intensity ( n = 5 mice per genotype per group, Scale bars: 200 μm). (D–F) Representative immunofluorescence analysis and quantification of total Mbp and oligodendrocytes <t>(Olig2</t> + ), and g-h) representative immunofluorescence analysis and quantification of mature oligodendrocytes (CC-1 + ) ( n = 5 mice per genotype per group, Scale bars: 50 μm). Data are presented as the mean ± SD. two-way ANOVA with Bonferroni multiple comparisons test in (C, E, F, and H); ∗∗ p < 0.01 and ∗∗∗ p < 0.001; ns, not significant compared with the Aurkb fl/fl group.
Anti Oligodendrocyte Transcription Factor 1, supplied by Rockland Immunochemicals, used in various techniques. Bioz Stars score: 86/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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Average 86 stars, based on 1 article reviews
anti oligodendrocyte transcription factor 1 - by Bioz Stars, 2026-04
86/100 stars
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dnas  (OriGene)
90
OriGene dnas
Aurkb deficiency in microglia disrupts remyelination and oligodendrocyte density in the CPZ-induced demyelination model (A) Adult Aurkb fl/fl and Cx3cr1 CreERT2/+ Aurkb fl/fl mice (2–3 months old) were i.p. injected with TAM for 5 consecutive days. After one month, the mice were fed a 0.25% CPZ-enriched diet for 5 weeks to induce demyelination (CPZ group) or followed by a CPZ-free normal diet for an additional 2 weeks (Recovery group). The corpus callosum (CC) was analyzed by immunofluorescence (IF) and Black-Gold II myelin staining. (B and C) Representative Black-Gold II myelin staining images and quantification of myelin intensity ( n = 5 mice per genotype per group, Scale bars: 200 μm). (D–F) Representative immunofluorescence analysis and quantification of total Mbp and oligodendrocytes <t>(Olig2</t> + ), and g-h) representative immunofluorescence analysis and quantification of mature oligodendrocytes (CC-1 + ) ( n = 5 mice per genotype per group, Scale bars: 50 μm). Data are presented as the mean ± SD. two-way ANOVA with Bonferroni multiple comparisons test in (C, E, F, and H); ∗∗ p < 0.01 and ∗∗∗ p < 0.001; ns, not significant compared with the Aurkb fl/fl group.
Dnas, supplied by OriGene, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/result/dnas/product/OriGene
Average 90 stars, based on 1 article reviews
dnas - by Bioz Stars, 2026-04
90/100 stars
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mouse monoclonal mbp  (Boster Bio)
90
Boster Bio mouse monoclonal mbp
Aurkb deficiency in microglia disrupts remyelination and oligodendrocyte density in the CPZ-induced demyelination model (A) Adult Aurkb fl/fl and Cx3cr1 CreERT2/+ Aurkb fl/fl mice (2–3 months old) were i.p. injected with TAM for 5 consecutive days. After one month, the mice were fed a 0.25% CPZ-enriched diet for 5 weeks to induce demyelination (CPZ group) or followed by a CPZ-free normal diet for an additional 2 weeks (Recovery group). The corpus callosum (CC) was analyzed by immunofluorescence (IF) and Black-Gold II myelin staining. (B and C) Representative Black-Gold II myelin staining images and quantification of myelin intensity ( n = 5 mice per genotype per group, Scale bars: 200 μm). (D–F) Representative immunofluorescence analysis and quantification of total Mbp and oligodendrocytes <t>(Olig2</t> + ), and g-h) representative immunofluorescence analysis and quantification of mature oligodendrocytes (CC-1 + ) ( n = 5 mice per genotype per group, Scale bars: 50 μm). Data are presented as the mean ± SD. two-way ANOVA with Bonferroni multiple comparisons test in (C, E, F, and H); ∗∗ p < 0.01 and ∗∗∗ p < 0.001; ns, not significant compared with the Aurkb fl/fl group.
Mouse Monoclonal Mbp, supplied by Boster Bio, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/result/mouse monoclonal mbp/product/Boster Bio
Average 90 stars, based on 1 article reviews
mouse monoclonal mbp - by Bioz Stars, 2026-04
90/100 stars
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oligodendrocyte transcription factor 2  (OriGene)
92
OriGene oligodendrocyte transcription factor 2
Aurkb deficiency in microglia disrupts remyelination and oligodendrocyte density in the CPZ-induced demyelination model (A) Adult Aurkb fl/fl and Cx3cr1 CreERT2/+ Aurkb fl/fl mice (2–3 months old) were i.p. injected with TAM for 5 consecutive days. After one month, the mice were fed a 0.25% CPZ-enriched diet for 5 weeks to induce demyelination (CPZ group) or followed by a CPZ-free normal diet for an additional 2 weeks (Recovery group). The corpus callosum (CC) was analyzed by immunofluorescence (IF) and Black-Gold II myelin staining. (B and C) Representative Black-Gold II myelin staining images and quantification of myelin intensity ( n = 5 mice per genotype per group, Scale bars: 200 μm). (D–F) Representative immunofluorescence analysis and quantification of total Mbp and oligodendrocytes <t>(Olig2</t> + ), and g-h) representative immunofluorescence analysis and quantification of mature oligodendrocytes (CC-1 + ) ( n = 5 mice per genotype per group, Scale bars: 50 μm). Data are presented as the mean ± SD. two-way ANOVA with Bonferroni multiple comparisons test in (C, E, F, and H); ∗∗ p < 0.01 and ∗∗∗ p < 0.001; ns, not significant compared with the Aurkb fl/fl group.
Oligodendrocyte Transcription Factor 2, supplied by OriGene, used in various techniques. Bioz Stars score: 92/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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Average 92 stars, based on 1 article reviews
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rabbit monoclonal antibody to pla2g2a  (Boster Bio)
96
Boster Bio rabbit monoclonal antibody to pla2g2a
Fig. 6. SND alleviated myocardial inflammation caused by MI through modulating AA metabolism pathway (mean ± SD, n = 5). (A) Expressions of <t>sPLA2,</t> COX-1 and COX-2 in cardiac tissue were measured by Western blot. (B) Expressions of 15-LOX and 5-LOX were measured by Western blot. ##P < 0.01 and ###P < 0.001 vs sham group. *P < 0.05 and **P < 0.01 vs model group.
Rabbit Monoclonal Antibody To Pla2g2a, supplied by Boster Bio, used in various techniques. Bioz Stars score: 96/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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Average 96 stars, based on 1 article reviews
rabbit monoclonal antibody to pla2g2a - by Bioz Stars, 2026-04
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oligo 2  (Promega)
90
Promega oligo 2
Fig. 6. SND alleviated myocardial inflammation caused by MI through modulating AA metabolism pathway (mean ± SD, n = 5). (A) Expressions of <t>sPLA2,</t> COX-1 and COX-2 in cardiac tissue were measured by Western blot. (B) Expressions of 15-LOX and 5-LOX were measured by Western blot. ##P < 0.01 and ###P < 0.001 vs sham group. *P < 0.05 and **P < 0.01 vs model group.
Oligo 2, supplied by Promega, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/result/oligo 2/product/Promega
Average 90 stars, based on 1 article reviews
oligo 2 - by Bioz Stars, 2026-04
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oligo 2  (TATAA Biocenter AB)
90
TATAA Biocenter AB oligo 2
Fig. 6. SND alleviated myocardial inflammation caused by MI through modulating AA metabolism pathway (mean ± SD, n = 5). (A) Expressions of <t>sPLA2,</t> COX-1 and COX-2 in cardiac tissue were measured by Western blot. (B) Expressions of 15-LOX and 5-LOX were measured by Western blot. ##P < 0.01 and ###P < 0.001 vs sham group. *P < 0.05 and **P < 0.01 vs model group.
Oligo 2, supplied by TATAA Biocenter AB, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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Average 90 stars, based on 1 article reviews
oligo 2 - by Bioz Stars, 2026-04
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importin-β oligo2  (Oligos Etc)
90
Oligos Etc importin-β oligo2
Fig. 6. SND alleviated myocardial inflammation caused by MI through modulating AA metabolism pathway (mean ± SD, n = 5). (A) Expressions of <t>sPLA2,</t> COX-1 and COX-2 in cardiac tissue were measured by Western blot. (B) Expressions of 15-LOX and 5-LOX were measured by Western blot. ##P < 0.01 and ###P < 0.001 vs sham group. *P < 0.05 and **P < 0.01 vs model group.
Importin β Oligo2, supplied by Oligos Etc, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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Average 90 stars, based on 1 article reviews
importin-β oligo2 - by Bioz Stars, 2026-04
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invader oligo 2 (seq id no: 6) (ica oligo 2)  (Fasmac Co Ltd)
90
Fasmac Co Ltd invader oligo 2 (seq id no: 6) (ica oligo 2)
Fig. 6. SND alleviated myocardial inflammation caused by MI through modulating AA metabolism pathway (mean ± SD, n = 5). (A) Expressions of <t>sPLA2,</t> COX-1 and COX-2 in cardiac tissue were measured by Western blot. (B) Expressions of 15-LOX and 5-LOX were measured by Western blot. ##P < 0.01 and ###P < 0.001 vs sham group. *P < 0.05 and **P < 0.01 vs model group.
Invader Oligo 2 (Seq Id No: 6) (Ica Oligo 2), supplied by Fasmac Co Ltd, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/result/invader oligo 2 (seq id no: 6) (ica oligo 2)/product/Fasmac Co Ltd
Average 90 stars, based on 1 article reviews
invader oligo 2 (seq id no: 6) (ica oligo 2) - by Bioz Stars, 2026-04
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ordered oligo 2- (seq id no: 4)  (Oligos Etc)
90
Oligos Etc ordered oligo 2- (seq id no: 4)
Fig. 6. SND alleviated myocardial inflammation caused by MI through modulating AA metabolism pathway (mean ± SD, n = 5). (A) Expressions of <t>sPLA2,</t> COX-1 and COX-2 in cardiac tissue were measured by Western blot. (B) Expressions of 15-LOX and 5-LOX were measured by Western blot. ##P < 0.01 and ###P < 0.001 vs sham group. *P < 0.05 and **P < 0.01 vs model group.
Ordered Oligo 2 (Seq Id No: 4), supplied by Oligos Etc, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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pl 100 photoinitiator  (Esstech Inc)
90
Esstech Inc pl 100 photoinitiator
Fig. 6. SND alleviated myocardial inflammation caused by MI through modulating AA metabolism pathway (mean ± SD, n = 5). (A) Expressions of <t>sPLA2,</t> COX-1 and COX-2 in cardiac tissue were measured by Western blot. (B) Expressions of 15-LOX and 5-LOX were measured by Western blot. ##P < 0.01 and ###P < 0.001 vs sham group. *P < 0.05 and **P < 0.01 vs model group.
Pl 100 Photoinitiator, supplied by Esstech Inc, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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Image Search Results


Aurkb deficiency in microglia disrupts remyelination and oligodendrocyte density in the CPZ-induced demyelination model (A) Adult Aurkb fl/fl and Cx3cr1 CreERT2/+ Aurkb fl/fl mice (2–3 months old) were i.p. injected with TAM for 5 consecutive days. After one month, the mice were fed a 0.25% CPZ-enriched diet for 5 weeks to induce demyelination (CPZ group) or followed by a CPZ-free normal diet for an additional 2 weeks (Recovery group). The corpus callosum (CC) was analyzed by immunofluorescence (IF) and Black-Gold II myelin staining. (B and C) Representative Black-Gold II myelin staining images and quantification of myelin intensity ( n = 5 mice per genotype per group, Scale bars: 200 μm). (D–F) Representative immunofluorescence analysis and quantification of total Mbp and oligodendrocytes (Olig2 + ), and g-h) representative immunofluorescence analysis and quantification of mature oligodendrocytes (CC-1 + ) ( n = 5 mice per genotype per group, Scale bars: 50 μm). Data are presented as the mean ± SD. two-way ANOVA with Bonferroni multiple comparisons test in (C, E, F, and H); ∗∗ p < 0.01 and ∗∗∗ p < 0.001; ns, not significant compared with the Aurkb fl/fl group.

Journal: iScience

Article Title: Aurkb deficiency disrupts microglial development, homeostasis and hinders remyelination following cuprizone-induced demyelination

doi: 10.1016/j.isci.2026.114718

Figure Lengend Snippet: Aurkb deficiency in microglia disrupts remyelination and oligodendrocyte density in the CPZ-induced demyelination model (A) Adult Aurkb fl/fl and Cx3cr1 CreERT2/+ Aurkb fl/fl mice (2–3 months old) were i.p. injected with TAM for 5 consecutive days. After one month, the mice were fed a 0.25% CPZ-enriched diet for 5 weeks to induce demyelination (CPZ group) or followed by a CPZ-free normal diet for an additional 2 weeks (Recovery group). The corpus callosum (CC) was analyzed by immunofluorescence (IF) and Black-Gold II myelin staining. (B and C) Representative Black-Gold II myelin staining images and quantification of myelin intensity ( n = 5 mice per genotype per group, Scale bars: 200 μm). (D–F) Representative immunofluorescence analysis and quantification of total Mbp and oligodendrocytes (Olig2 + ), and g-h) representative immunofluorescence analysis and quantification of mature oligodendrocytes (CC-1 + ) ( n = 5 mice per genotype per group, Scale bars: 50 μm). Data are presented as the mean ± SD. two-way ANOVA with Bonferroni multiple comparisons test in (C, E, F, and H); ∗∗ p < 0.01 and ∗∗∗ p < 0.001; ns, not significant compared with the Aurkb fl/fl group.

Article Snippet: Rabbit anti-Olig2 Antibody , Proteintech , Cat# 13999-1-AP, RRID: AB_2157541.

Techniques: Injection, Immunofluorescence, Staining

Fig. 6. SND alleviated myocardial inflammation caused by MI through modulating AA metabolism pathway (mean ± SD, n = 5). (A) Expressions of sPLA2, COX-1 and COX-2 in cardiac tissue were measured by Western blot. (B) Expressions of 15-LOX and 5-LOX were measured by Western blot. ##P < 0.01 and ###P < 0.001 vs sham group. *P < 0.05 and **P < 0.01 vs model group.

Journal: Chinese Herbal Medicines

Article Title: Sini decoction alleviates inflammation injury after myocardial infarction through regulating arachidonic acid metabolism

doi: 10.1016/j.chmed.2023.12.004

Figure Lengend Snippet: Fig. 6. SND alleviated myocardial inflammation caused by MI through modulating AA metabolism pathway (mean ± SD, n = 5). (A) Expressions of sPLA2, COX-1 and COX-2 in cardiac tissue were measured by Western blot. (B) Expressions of 15-LOX and 5-LOX were measured by Western blot. ##P < 0.01 and ###P < 0.001 vs sham group. *P < 0.05 and **P < 0.01 vs model group.

Article Snippet: The primary antibodies including rabbit monoclonal antibody to 5 Lipoxygenase (ab169755, Abcam, Boston, MA, USA), rabbit monoclonal antibody to 15 Lipoxygenase (ab244205, Abcam, Boston, MA, USA), rabbit monoclonal antibody to NF-kB p65 (ab32536, Abcam, Boston, MA, USA), rabbit monoclonal antibody to COX-1 (ab109025, Abcam, Boston, MA, USA), rabbit monoclonal antibody to PLA2G2A (M02259-1, BOSTER, Wuhan, China), rabbit monoclonal antibody to COX-2 (66351-1-1 g, Proteintech, Wuhan, China), rabbit polyclonal antibody to Bax (50599-2-1 g, Proteintech, Wuhan, China), rabbit polyclonal antibody to Bcl-2 (26593- 1-AP, Proteintech, Wuhan, China), rabbit polyclonal antibody to Cleaved Caspase-3 (19677-1-AP, Proteintech, Wuhan, China), rabbit polyclonal antibody to TNF alpha (17590-1-AP, Proteintech, Wuhan, China), rabbit polyclonal antibody to IL-6 (DF6087, Affinity Biosciences, Cincinnati, Ohio, USA), rabbit polyclonal antibody to Beta-actin (GB11001, Servicebio, Wuhan, China).

Techniques: Western Blot